In tomorrow's May edition of Science, researchers from Harvard Medical school and University of Wisconsin-Madison are reporting a new discovery regarding the molecular underpinnings of CMV host infections.

The scientists found that CMV mimics a human host cell protein to help hijack the cell's machinery. The viral protein UL57 mimics a human protein and functions as a regulator of a tumor suppressor gene. The viral protein modifies the tumor suppressor gene Rb to accelerate cell growth which is preferable for the virus's proliferation.

Check out the original article here.

yours in calici,

Lauren